![]() ![]() Please measure yourself before casting on to estimate which size will fit you best. Therefore, the sizes XS (S) M (L) XL correspond to a bust measurement of approx. This intended positive ease is built into the pattern. The Scotty Sweater is meant to be worn with a positive ease of approx. Knit a swatch to find the needle size that gives you the correct gauge. The decorative lines for the characteristic pattern on the Scotty Sweater are embroidered at the end using duplicate stitches. The Scotty Sweater requires almost no finishing, except for sewing down the folded neckband and the cuffs at the sleeves. The neckband is knit in rib from picked-up stitches along the neckline and is folded over for a double collar. Next, you will pick up stitches along the armholes for working the set-in sleeves using short rows. You will begin by knitting the top back, then the shoulders, the front, and then the body of the sweater. You can substitute for another yarn or yarn combination that gives the same gauge, but keep in mind that the fabric characteristics may vary. This is a further gamble, because stranded colorwork (as Naomi told me on Twitter) tends to have square stitches, and intarsia has rectangular ones.The Scotty Sweater is knit from the top down in stockinette stitch holding one strand of Spinni and two strands of Silk Mohair together. My plan is to further simplify the intarsia chart down to a stranded colorwork chart. ![]() ![]() I’d enjoy it if you sent me a photo though, so I can cheer you on. If you want to use this chart on your knitting, or the twist stitch one, be my guest. It will join other swatches in a bag until there are enough to piece into a blanket. If you don’t count the 3 times I picked out the embroidery, this owl has the honor of being knit once from the first chart. My husband and sons are always telling me to use more color in my knitting, so this swatch had a happy reception. The embroidery took longer than the knitting did, because I cannot count! I actually had to pick out my embroidery 3 times. I love it when the computer does fiddly things for me. Then the program re-colors the sections for you. a question box will jump up asking which color you wish to replace it with. One feature of Stitchmastery that makes simplifying colorwork charts easier is that if you are, say turning all of your midtone brown into pale brown, you can highlight the midtone brown in the key, and delete it. The more references for embroidery the less I get lost. I was happy with how it turned out, so I’m not going to re-do it, but if I were, I’d knit in the white patches in the tail, body and edges of the face. Once I had knit the intarsia base, I duplicate stitched the details on it.Īctually the first chart I’d made had the details, and I simplified it to the intarsia chart above. I was gambling that the proportions would work from twist stitch to intarsia, since twist stitches pull in, and intarsia does not. One had an owl sitting in a pine tree, which was great because the green yarn that I had went with the brown yarn better than the blue yarn that I had. I did monkey with the colors a bit, it turns out there are color variations in Northern Saw Whets, so I looked at reference photos on Google Images until I found a pretty one with colors like yarns I had in leftovers or stash. I decided to do the intarsia owl next because I found a chart on my hard drive all ready to go, based on the twist stitch owl. All these interviews have made me ready to make the next owl.
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It's actually happened twice against the Bucs - in 1999, the Vikings got three touchdowns from Randall Cunningham (two to Randy Moss) and led the Bucs 21-0 after one quarter, holding on for a 21-14 win. Arizona won 14-6 against the Lions in 2014, and that's the only other time it's happened since the start of the 2013 season. In the last 30 years - that's roughly 7,500 games - it's happened just nine times. The Bucs generally don't get shut out like that - since the start of the 2014 season, Tampa Bay only had one other game where they failed to score after the first quarter - this year's 27-7 loss to the Broncos.Īcross the NFL, it's fairly rare to not score after the opening period and still win. Since then, 26 times it's happened, all Bucs losses by at least 14. To find a game in which the Bucs didn't score after the first quarter and even lost by less than 14 points, you have to go clear back to 1984 and a 13-7 overtime loss to the Lions. The one other time they had a 14-0 lead after the first quarter and failed to score the rest of the game, more predictably, the Bucs lost 32-14 to the Patriots in 1985. They were 0-11 in their first two seasons alone - all shutout losses. ![]() ![]() This is hardly a recipe for NFL success - the Bucs jumped out to a 14-0 lead in the opening quarter, then played well enough on defense to pull off a 14-5 win over the Seattle Seahawks.īefore Sunday, when the Bucs had failed to score a single point after the first quarter of a game, they had been 0-44 all-time. The Bucs won on Sunday despite not scoring a single point after the first quarter. It's always impressive that the NFL plays 256 games in every regular season and you can still find things that don't happen for years at a time, or even once in the 41-year history of a franchise. ![]() Measurement of 17-hydroxyprogesterone (17-OHP) with or without ACTH stimulation establishes the diagnosis of CAH due to 21-OHD. Nonclassic: 46,XY variable degrees of genital ambiguity, adrenal insufficiency.Ĭholesterol side-chain cleavage enzyme deficiencyĬlassic: phenotypic female (46,XX or 46,XY sex reversal), adrenal insufficiency, salt-wasting.Ībbreviations: CAH, congenital adrenal hyperplasia CAH-X, congenital adrenal hyperplasia with tenascin-X impairment StAR, steroidogenic acute regulatory protein Post-natal virilization does not occur.Ĭlassic: phenotypic female (46,XX or 46,XY sex reversal), adrenal insufficiency, severe salt-wasting Possible skeletal malformations (Antley-Bixler syndrome). Partial: 46,XY variable degrees of genital ambiguity.Ĥ6, XX variable development of secondary sexual characteristics.ģβ-hydroxysteroid dehydrogenase type 2 deficiencyĬlassic: 46,XX and 46,XY ambiguous genitalia, adrenal insufficiency, salt-wasting.Ĥ6,XX and 46,XY ambiguous genitalia, adrenal insufficiency, severe salt-wasting, possible maternal virilization during pregnancy. May be asymptomatic.Ĭlassic: female phenotype (46,XX or 46,XY sex reversal), hypertension, pubertal delay with absence of secondary sexual characteristics. Nonclassic: hyperandrogenism during childhood or early adulthood. Nonclassic: hyperandrogenism during childhood or early adulthood may be asymptomatic.ĬAH-X: In addition to the above, joint hypermobility, joint pain, multiple joint dislocations, midline defects including possible cardiac structural abnormalities.Ĭlassic: 46,XX ambiguous genitalia, postnatal virilization, hypertension. 4 Other less common forms of CAH include: 3β-hydroxysteroid dehydrogenase type 2 deficiency, 17α-hydroxylase deficiency that is more commonly seen in Mennonite descendants of Dutch Frieslander and the Brazilian population congenital lipoid adrenal hyperplasia that is more commonly seen in the Japanese and Korean populations side-chain cleavage enzyme deficiency that is most commonly found in Turkey and cytochrome P450 oxidoreductase deficiency (ORD), the only variant that can manifest with skeletal malformation.Ĭlassic: 46,XX ambiguous genitalia, adrenal insufficiency, salt-wasting, postnatal virilization. 3 The second most common form of CAH, 11-hydroxylase deficiency (11-OHD), occurs in 1 in 100,000 live births in the general population and accounts for approximately 5% of cases. 2 NCCAH is one of the most common autosomal recessive disorders in humans and affects approximately 1 in 1,000 individuals. 1 The classic type affects approximately 1 in 16,000 live births. 21-OHD is classified into 3 subtypes according to clinical severity: classic salt-wasting (SW), classic simple virilizing (SV), and nonclassic (NCCAH, mild or late-onset). Over 95% of all cases of CAH are caused by 21- hydroxylase deficiency (21-OHD). The severity and clinical features of CAH vary depending on the enzymatic defect, its residual activity, age of presentation, and genotype.ĬAH represents a continuous phenotypic spectrum ( Table 1). The biochemical defects in CAH translate to a spectrum of clinical consequences, which include adrenal insufficiency, genital ambiguity or disordered sex development (DSD), infertility, short stature, hypertension, and an increased risk of metabolic syndrome during adolescence and adulthood. This leads to defective cortisol synthesis, shunting of the accumulated steroid precursors through alternative pathways, and often adrenal gland hyperplasia. Consequently, overproduction of corticotropin releasing hormone (CRH) and adrenocorticotropic hormone (ACTH) from the hypothalamus and pituitary glands, respectively, results in an increase and accumulation of various steroid precursors proximal to the block. In this article, we provide an in-depth discussion on the genetics of CAH, including genetic diagnosis, molecular analysis, genotype-phenotype relationships and counseling of patients and their families.Ĭongenital adrenal hyperplasia (CAH) refers to a group of autosomal recessive disorders that impair cortisol biosynthesis. Several pitfalls in the genetic diagnosis of patients with CAH exist. Certain ethnic groups have a predilection to certain genotypes, which may have resulted from an ancient founder effect, a hot spot in the gene, unequal crossing over during meiosis or gene conversion of point mutations from a pseudogene. The genes for the various variants of CAH are well characterized, and mutation analysis is widely available. ![]() Genotyping is an important tool in confirming the diagnosis or carrier state, provides prognostic information on disease severity, and is essential for genetic counseling. ![]() CAH represents a continuous phenotypic spectrum with over 95% of all cases caused by 21-hydroxylase deficiency. Congenital adrenal hyperplasia (CAH) refers to a group of autosomal recessive disorders due to single gene defects in the various enzymes required for cortisol biosynthesis. ![]() Play the complete classic game with no pay-to-win or ad pop-ups The video chat feature is optional and you can easily opt out. If you want to chat while you play, create an account, add friends, start games from your group chats and move automatically to video when you begin. ![]() Play the Hasbro classic MONOPOLY game by yourself, with family and friends or players around the world on your mobile or tablet! 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On the other hand, he wears blue overalls, which are extremely dirty, and has brown hair, so he is very lazy, which will lower his intelligence. For example, many of the foods and drinks SpongeBob eats are washed down by the sea, so this will lower his health. Although it is a game of luck, there are also other factors such as health, clothing, and intelligence that will affect whether you win or lose your life. One of the greatest things about SpongeBob Squarepants is how open its rules are. This game had a lot of success and was a huge hit among early Americans, which is why it became a board game for the US. The Game of Life required players to make color choices and then see if they "paid" by moving their colored pieces around the play field. It can be credited for starting the party game night that we know today as a time when parties were a gathering of families and friends. The Game of Life is the most popular of all board games, and also the most popular parlor game in America. SpongeBob SquarePants - The Game of life is an abstract board game, originally developed in 1860 by Milton Bradley. |
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